slypher
100+ Head-Fier
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I take my meter to the lab with me every once in a while to have its acuracy verified. Its an easy way to confirm your test results
Originally Posted by amphead /img/forum/go_quote.gif They need to make a commitment to annihilate that damned disease, before it gets everybody. Seems like more and more have been diagnosed and the numbers just seem to be growing at an alarming rate. It's probably something in the water or the soil, and the human body is rebelling. |
Originally Posted by billybob_jcv /img/forum/go_quote.gif Also, it's worth noting that there are 2 types of blood glucose test results - plasma and whole blood. Whole blood readings are ~12% lower than plasma readings. Historically, lab results were given as plasma, while most home meters gave whole blood readings. IMHO this led to much of the confusion about the "inaccuracy" of home meters. But, that has changed. I think all the major home meters now give plasma readings, making them easier to compare to lab results. But - I seem to recall reading that ALL home meters are considered "accurate" if they are within 20% of lab readings. So, a lab reading of 100 mg/dL might show up as anywhere between 80 & 120 on a home meter! Finally, the meter in the doctor's office is NOT a lab test - it's just a meter made tougher for constant use. A "real" plasma glucose test involves centrifuging to separate the plasma - so comparing the lab test results to the doctor's meter would also show a significant difference. FYI - IANAD, YMMV, etc... |
Originally Posted by simpleworld /img/forum/go_quote.gif If a diabetic person is either insulin resistant or does not produce insulin at all, wouldn't the best treatment be to eliminate carbohydrates from the diet? Last I checked, carbs are in no way required in the human diet. Has anyone considered that Type 1 diabetes can come about in the womb of a mother who eats a high carb diet while pregnant, thus taxing the babies pancreas from before birth? |
Originally Posted by DanTheMiataMan /img/forum/go_quote.gif So then the body feels as it is starving as it is not getting what it needs from the blood. So then it starts to slowly turn to a negative PH so it can break down fat. Once all your fat is broken down it next moves to muscle to break down because it has been starving for so long. As all the fat it broke down could still not be used very well or at all by your body without insulin. This is called Ketone Acidosis (sp) and is a life ender for diabetics that can not keep their blood sugars in controll. As once it turns to a negative enough PH you have massive body wide organ failure. |
Originally Posted by amphead /img/forum/go_quote.gif They need to make a commitment to annihilate that damned disease, before it gets everybody. Seems like more and more have been diagnosed and the numbers just seem to be growing at an alarming rate. It's probably something in the water or the soil, and the human body is rebelling. |
Originally Posted by DNA Doc /img/forum/go_quote.gif Actually, for the most part, the increases in incidence and prevalence are due to people eating too much and moving too little. A little lifestyle modification goes a long way here. |
Originally Posted by Drag0n /img/forum/go_quote.gif I guess i can assume you do not have a One Touch meter, because youre very strong on this subject. Buy what you wish. |
Originally Posted by simpleworld /img/forum/go_quote.gif If a diabetic person is either insulin resistant or does not produce insulin at all, wouldn't the best treatment be to eliminate carbohydrates from the diet? Last I checked, carbs are in no way required in the human diet. Has anyone considered that Type 1 diabetes can come about in the womb of a mother who eats a high carb diet while pregnant, thus taxing the babies pancreas from before birth? |
Originally Posted by paara /img/forum/go_quote.gif Hello I can se that you have a large knowledge about DM and isulin, but here are some additional information about ketoacidosis Hope it is not inapropriate and to much off topic. - Diabetic ketoacidosis: It is almost exlusivly seen in DM1. It is caused by profound insulin loss. There is so little insulin that adipose tissue start to really catabolize lipids without controll, providing fatty acids for production of ketons. (Fatty tissue is the organ whis has the highest sensitivity to insulin, and it lose controlly in severe loss of insulin, such as seen in DM1, and not DM2) This fat is released into the blood and converted into ketons or back to triglycerides by the liver There is typically severe hyperglycemia, which cause hyperosmolarity of the plasma. Initially this is compensated for by shifting of water out of the cell and the patient has polydipsia to compensate for the polyuria. But because of nausea and vomiting as a result of the beginning ketoacidosis this mechanism fail and the depleted intravascular volume leads to decreased bloodflow to the kidneys. (it is the hypovolemia which is the reason for coma and death in most cases, not the acidosis, which many think) Hypovolemia activates stress mediators counteract the little insulin left. (such as adrenalin, glucagon, corticosteroids etc. ) Glucose levels rise abruptly because it is not lost through the kidney any more (because of hypovolemia, blood is shunted away from the kidney. The normal protectiv mechanism by loosing glucose through the urine is removed, and glucose increase uncontrollaby). Coma occur as a result of the hyperosmolarity and loss on intracellular volume. Keton bodies are acids that lower pH. Causing some of the symptoms of ketoacidosis such as kussmal breathing (rapid deep breathing) Na+ is lost from the polyuria, so is K+, but because of the low pH, K+ shifts out from the cells and severe hypokalemia do not develop. But when administrating insulin glucose pull with it K+ back in the cells and severe hypokalemia develops. This can cause arrhythmias and death. - Nonketoic hyperosmolar coma: usually develop in DM 2 in the setting of severe dehydration. Because of the absence of ketoacidosis and its symptoms patients often presents later, and therefor have more profound hyperglycemia and dehydration. Mortality is 10x higher then for ketoacidosis. |